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Activated platelets produce a compound called thromboxane, which causes them to become sticky and recruits neighboring platelets to form a mesh, which ultimately becomes a blood clot.

Aspirin blocks an enzyme within platelets that makes thromboxane.

NSAIDs reduce pain and inflammation by preventing the production of prostaglandins and thromboxane, chemical compounds that cause blood vessels to constrict.

Prostaglandins and thromboxane are synthesized from a fatty substance in the brain and muscle tissue that is involved in cellular repair.

Antiplatelets such as aspirin, dipyridamole, ticlopidine and clopidogrel prevent clotting by blocking thromboxane, a chemical that rounds up clotting cells in the blood.